Programs

Five areas of study

Each course stands on its own, and all five together make up the coursework for the full certification. Courses run $495 to $795 each, and tuition is credited if you go on to certify.

Every course is built the same way: the mechanism first, in plain words, then the practice, then how you'd know if it's working for a real person. We teach what the evidence supports and we flag where it runs thin.

I.

Ancestral Bioavailable Nutrients

A nutrient on a label isn't a nutrient in your blood. Heme iron from red meat is absorbed at roughly 15 to 35 percent; the non-heme iron in plants, at 2 to 20 percent, and less when phytates are in the same meal. Liver carries vitamin A as ready-made retinol, while the beta-carotene in carrots has to be converted, and how well people convert it varies a lot from one person to the next. B12 is found naturally only in animal foods; plants have none unless it's added. Ready-made DHA and EPA come mainly from fish and seafood, with smaller amounts in eggs and pasture-raised meat; the body turns only a small fraction of plant ALA into them.

You'll learn how to build a week of meals around meat, fat, eggs, fish and organs that people will actually eat, how to source and cook liver and heart so they're not a punishment, and how to read a blood panel with a client's doctor in mind.

II.

Connected Circadian Practices

Light-sensitive cells in the retina (the melanopsin cells, or ipRGCs) report brightness straight to the suprachiasmatic nucleus, the brain's master clock. An overcast morning outside is often ten thousand lux or more; a lit kitchen is a few hundred. That difference is what tells the clock it's morning, which times the cortisol rise after waking and, about fourteen to sixteen hours later, the release of melatonin. Bright light late at night pushes that melatonin back.

Meals are a second clock signal for the liver and gut. The course covers morning light routines for people who move slowly in the morning, how to make a bedroom properly dark, when to eat the last meal, and how to track sleep without becoming a slave to a ring or a watch.

III.

Struggle Reward Pathways Flagship

Dopamine isn't a pleasure chemical. It's closer to a teaching signal: it fires when a result beats what you expected and drops when it falls short, and it sets how much effort a goal feels worth. Parkinson's is a loss of dopamine-producing cells, which is why low motivation and "I'll do it later" are part of daily life for so many of our members. Apathy, a real loss of drive, affects roughly 4 in 10 people with Parkinson's, and it's tied to the same dopamine loss. That's why this course is the center of the program.

The course is about structuring hard things a person chooses for themselves: a fast held to the hour they set, a hill walked every morning, a heavy set done with good form. Each is effort now and reward later. Done on a schedule, our aim is to get the reward system firing on work and waiting instead of on the next easy hit. That's our coaching model, not a proven brain effect. You'll learn how to size a challenge so it's hard but finishable, how to log it so the win is visible, and how to coach someone through the flat days when nothing feels worth it.

Our view is old-fashioned: meaning comes from carrying something heavy on purpose, and nobody can carry it for you. A coach can set the weight and stand beside you.

IV.

Autophagy and Lipid Interchange

When you stop eating, insulin falls and glucagon rises. The liver works through its glycogen over roughly the first day, fat cells release fatty acids, and the liver turns some of them into ketones, mainly beta-hydroxybutyrate. In blood, a BHB reading of 0.5 mmol/L or higher is the usual line for nutritional ketosis. In the same low-insulin, low-nutrient state, AMPK rises and mTOR quiets down, which is the switch that lets cells start autophagy: breaking down worn-out proteins and damaged parts and reusing the material.

The lipid interchange half is about where fat actually goes. When fat is burned, most of its mass leaves the body as carbon dioxide you breathe out (about 84 percent by one well-known calculation) and the rest as water.

Nobody can measure autophagy with a home test, and we don't pretend to. What we can check is ketosis, the fasted state where, in lab and animal research, autophagy turns up. In people we can't measure it directly yet, so we count the hours instead of claiming a number, and bank them on the Autophagy Ledger: a lifetime balance that never resets. A urine strip reads a different ketone (acetoacetate), not the BHB a blood meter reads. A pink strip tells you you're burning fat for fuel. A blank one doesn't prove you're out of ketosis, especially once you're fat-adapted. A blood meter is the more exact check. The course covers fasting schedules, electrolytes, breaking a fast well, who should not fast without their prescriber involved, and how to read strips honestly.

V.

Optimal Body Composition and Hormonal Practices

Skeletal muscle is the biggest single place blood sugar goes after a meal, so muscle you keep is blood sugar you control. Muscle protein synthesis needs both a reason (load) and the raw material (enough protein per meal, with roughly 2.5 to 3 grams of leucine as a practical threshold). Fasting raises growth hormone, which helps protect lean tissue while fat is used for fuel. Poor sleep pushes cortisol and appetite up and testosterone down.

You'll learn how to program strength work for people who are older or unsteady, how to set protein targets on a carnivore or ketogenic plan, and how to measure progress without lying to yourself: waist-to-height ratio, grip strength, the 30-second chair stand, and a DEXA scan where one is available.

Dates and format

Course dates and lengths are being set with the first cohort. If you want to hear when enrollment opens, use the form on the Certification page.